DR MANISH-2.pdf laser proctology piles and fistula

Jan 27, 20250 likes18 views

Get the best piles and fistula treatment in Jaipur with advanced laser proctology techniques. Dr. Manish Rajput, a renowned specialist, provides painless, quick, and highly effective treatments for piles, fissures, and fistula. Using cutting-edge laser technology, the procedures ensure minimal discomfort, no stitches, and faster recovery, helping patients return to their daily lives with ease. Dr. Rajput is committed to offering personalized care and long-term solutions, ensuring lasting relief and improved quality of life. With his expertise and modern facilities, you can expect safe and reliable treatment for all proctological conditions.

D R M A N I S H
RAJPUT
ht t ps://dr manishr ajput .com
Bookan appointment!

IN T R O D U C T IO N
Dr
. Manish Rajput is an I
nterventional
Radiologist & Team Lead, Team I
R
Jaipur
. They are the biggest team of
I
nterventional Radiologists. They are
trained from Tata Memorial Center
,
Mumbai, I
ndia. They have worked in so
many government and corporate
hospitals across the country.

Medical school (MBBS):2005-2011: -People’s
Medical College, Bhopal(MP)
DNB (Radio diagnosis):
- Apollo hospital,
Hyderabad(Telangana)
FVIR (PDCC):- Tata Memorial Centre,
Mumbai(Maharashtra)
Senior Resident: Hinduja Hospital Mumbai, SMS
Hospital Jaipur
Past Visiting Doctor:Leelavati Hospital Mumbai,
Breach Candy Hospital Mumbai, Wockhardt
Hospital Mumbai, Hinduja Hospital Mumbai
Ex Assitant Professor:JNU Medical College, Jaipur
Currently Working as Senior Consultant
Interventional Radiologist in various corporate
hospitals of Rajasthan based in Jaipur
HIS
EDUCATION

S T R E N G T H S
Ilead the biggest I
R team in the state.
Vast portfolio for I
R services.
All the team members are from Tata
Memorial Hospital, Mumbai.
Extensive experience in performing and
interpreting basic Radio-Diagnosis.
Gained experience in performing
I
nterventional Radiologic procedures.
Ipossess oratory skill by speaking at
numerous industry events.
Ability to teach complex concepts in a basic
manner
.

Varicose Veins Prostate Artery Embolization PRG
Biopsy and
fNAC
Angioplasty & Venoplasty PCN & DJ Stenting
O
U
R
S
E
R
V
I
C
E
S

+91 7729021111
dr.manish@infinityintervention.com
O-5-A, Adinath Marg, Near Surya
Hospital, C Scheme, Ashok Nagar,
Jaipur, Rajasthan 302001
C ON TA C T
US!

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TRACHEOESOPHAGEAL FISTULA.pdf FOR NURSING STUDENTSKIRAN KARETHA

9 slides•141 views

diabetes mcq by NAME ANKUSH GOYAL (1).pdf by Dr Ankush goyal, has 14 slides with 159 views.Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose

diabetes mcq by NAME ANKUSH GOYAL (1).pdfDr Ankush goyal

14 slides•159 views

Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose

Cosmetic Dentistry in Birmingham – Transform Your Smile.pptxRobinhood Dental Practice

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Psychopharmacology questions .pdf by Dr ankush goyal by Dr Ankush goyal, has 17 slides with 168 views.Psychopharmacology: A Comprehensive Overview Introduction Psychopharmacology is the scientific study of the effects of drugs on mood, perception, cognition, and behavior. It involves the interaction of drugs with the nervous system, particularly the brain, to alter mental functions and treat psychiatric disorders. Psychopharmacology integrates principles from neuroscience, pharmacology, psychiatry, and psychology to develop effective treatments for mental illnesses such as depression, schizophrenia, anxiety, and bipolar disorder. History and Development The field of psychopharmacology has its roots in ancient medicine, where herbal remedies were used to treat mental disorders. Modern psychopharmacology began in the 20th century with the discovery of drugs such as chlorpromazine (antipsychotic) and imipramine (antidepressant), revolutionizing psychiatric care. Over time, research has led to the development of more targeted and effective drugs with fewer side effects. Neurotransmitter Systems and Drug Actions Psychotropic drugs exert their effects by modulating neurotransmitter systems in the brain. The major neurotransmitters involved include: Dopamine (DA): Associated with reward, motivation, and movement; implicated in schizophrenia and Parkinson’s disease. Serotonin (5-HT): Regulates mood, appetite, and sleep; targeted by antidepressants. Norepinephrine (NE): Involved in arousal and stress response; plays a role in depression and anxiety. Gamma-Aminobutyric Acid (GABA): The primary inhibitory neurotransmitter; targeted by anxiolytics and sedatives. Glutamate: The primary excitatory neurotransmitter; implicated in schizophrenia and cognitive function. Acetylcholine (ACh): Important for memory and learning; affected in Alzheimer’s disease. Classification of Psychotropic Drugs Psychotropic drugs can be classified based on their therapeutic use: 1. Antidepressants Used to treat depressive disorders, anxiety disorders, and other conditions. Selective Serotonin Reuptake Inhibitors (SSRIs): Fluoxetine, Sertraline, Citalopram Serotonin-Norepinephrine Reuptake Inhibitors (SNRIs): Venlafaxine, Duloxetine Tricyclic Antidepressants (TCAs): Amitriptyline, Imipramine Monoamine Oxidase Inhibitors (MAOIs): Phenelzine, Selegiline Atypical Antidepressants: Bupropion, Mirtazapine 2. Antipsychotics Used to manage schizophrenia, bipolar disorder, and psychotic disorders. Typical (First-Generation) Antipsychotics: Haloperidol, Chlorpromazine Atypical (Second-Generation) Antipsychotics: Risperidone, Olanzapine, Clozapine 3. Mood Stabilizers Used to treat bipolar disorder and mood dysregulation. Lithium: Effective for bipolar disorder Anticonvulsants: Valproate, Lamotrigine, Carbamazepine 4. Anxiolytics and Sedative-Hypnotics Used for anxiety and sleep disorders. Benzodiazepines: Diazepam, Lorazepam, Alprazolam Non-Benzodiazepine Hypnotics: Zolpidem, Eszopiclone Barbiturates: Phenobarbital (rarely used) Beta-Blockers: Propranolol

Psychopharmacology questions .pdf by Dr ankush goyalDr Ankush goyal

17 slides•168 views

Psychopharmacology: A Comprehensive Overview Introduction Psychopharmacology is the scientific study of the effects of drugs on mood, perception, cognition, and behavior. It involves the interaction of drugs with the nervous system, particularly the brain, to alter mental functions and treat psychiatric disorders. Psychopharmacology integrates principles from neuroscience, pharmacology, psychiatry, and psychology to develop effective treatments for mental illnesses such as depression, schizophrenia, anxiety, and bipolar disorder. History and Development The field of psychopharmacology has its roots in ancient medicine, where herbal remedies were used to treat mental disorders. Modern psychopharmacology began in the 20th century with the discovery of drugs such as chlorpromazine (antipsychotic) and imipramine (antidepressant), revolutionizing psychiatric care. Over time, research has led to the development of more targeted and effective drugs with fewer side effects. Neurotransmitter Systems and Drug Actions Psychotropic drugs exert their effects by modulating neurotransmitter systems in the brain. The major neurotransmitters involved include: Dopamine (DA): Associated with reward, motivation, and movement; implicated in schizophrenia and Parkinson’s disease. Serotonin (5-HT): Regulates mood, appetite, and sleep; targeted by antidepressants. Norepinephrine (NE): Involved in arousal and stress response; plays a role in depression and anxiety. Gamma-Aminobutyric Acid (GABA): The primary inhibitory neurotransmitter; targeted by anxiolytics and sedatives. Glutamate: The primary excitatory neurotransmitter; implicated in schizophrenia and cognitive function. Acetylcholine (ACh): Important for memory and learning; affected in Alzheimer’s disease. Classification of Psychotropic Drugs Psychotropic drugs can be classified based on their therapeutic use: 1. Antidepressants Used to treat depressive disorders, anxiety disorders, and other conditions. Selective Serotonin Reuptake Inhibitors (SSRIs): Fluoxetine, Sertraline, Citalopram Serotonin-Norepinephrine Reuptake Inhibitors (SNRIs): Venlafaxine, Duloxetine Tricyclic Antidepressants (TCAs): Amitriptyline, Imipramine Monoamine Oxidase Inhibitors (MAOIs): Phenelzine, Selegiline Atypical Antidepressants: Bupropion, Mirtazapine 2. Antipsychotics Used to manage schizophrenia, bipolar disorder, and psychotic disorders. Typical (First-Generation) Antipsychotics: Haloperidol, Chlorpromazine Atypical (Second-Generation) Antipsychotics: Risperidone, Olanzapine, Clozapine 3. Mood Stabilizers Used to treat bipolar disorder and mood dysregulation. Lithium: Effective for bipolar disorder Anticonvulsants: Valproate, Lamotrigine, Carbamazepine 4. Anxiolytics and Sedative-Hypnotics Used for anxiety and sleep disorders. Benzodiazepines: Diazepam, Lorazepam, Alprazolam Non-Benzodiazepine Hypnotics: Zolpidem, Eszopiclone Barbiturates: Phenobarbital (rarely used) Beta-Blockers: Propranolol

Adopting Modern Management Strategies for Obesity: Treating the Patient, Not ...PVI, PeerView Institute for Medical Education

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Co-Chairs and Presenters, Professor Carel le Roux, MBChB, FRCP, FRCPath, PhD, and Donna H. Ryan, MD, FTOS, discuss obesity in this CME activity titled “Adopting Modern Management Strategies for Obesity: Treating the Patient, Not the Disease.” For the full presentation, downloadable Practice Aids, and complete CME information, and to apply for credit, please visit us at https://bit.ly/40HfQWa. CME credit will be available until March 27, 2026.

PRETERM LABOR.pptx FOR NURSING STUDENTS CREATED BY KIRAN KARETHA by KIRAN KARETHA, has 40 slides with 52 views.PREMATURE LABOUR/ PRETERM LABOUR DEFINITION:  Pre term labour is defined by WHO as onset of labour prior to the completion of 37 weeks of gestation in a pregnancy beyond 20 weeks of gestation. INCIDENCE:  Globally, approximately 1 in 10 babies are born preterm (before 37 completed weeks of gestation), with an estimated 13.4 million preterm births annually. This means the incidence of preterm birth is roughly 10-11% worldwide.  In India, about 12% of babies are born preterm, which is higher than neighboring countries. ETIOLOGY:  In about 50%, the cause of preterm labour is not known- IDIOPATHIC  Some of the high-risk factors are:  HISTORY: previous history of abortion or preterm delivery Recurrent UTI Smoking habits Low socio-economic & nutritional status Previous abortion history Malpresentation  COMPLICATIONS IN PRESENT PREGNANCY: it may be due to maternal, fetal and placental. SIGN AND SYMPTOMS:  Backache  Contractions every 10 minutes, are more often  Cramping in lower abdomen  Menstrual like cramps  Fluid leaking from vagina  Flu like symptoms  Increased pressure in pelvis  Increased vaginal bleeding  Regular uterine activity  Vaginal spotting DIAGNOSIS:  Regular uterine contractions with or without pain (at least one in every 10 minutes.)  Dilation (2 cm or more) & effacement (80%) of the cervix  Length of cervix (2.5 cm or more.)  Funneling of internal OS INVESTIGATIONS:  Blood test  Urine analysis, urine culture and sensitivity  Cervicovaginal swab: culture and fibronectin test  Serum electrolyte and glucose level  USG COMPLICATION:  Birth of a pre-term baby A pre-term baby usually has following problems: • Low birth weight • Birth asphyxia • Neonatal jaundice • Underdeveloped organs etc. PREVENTIVE MEASURES: However, it is not possible to prevent occurrence of preterm labour completely, though its prevalence can be reduced using various measures at following three levels- 1) Prevention at primary level 2) Prevention at secondary level 3) Prevention at tertiary level 1) Prevention at primary level  In this, actions are taken prior to the onset of any disease which reduce or remove the possibility for occurring a disease.  For preventing preterm labour, following measures are applied at primary level. • Encourage the pregnant, to visit antenatal clinic regularly. • To identify high risk factors at its incipient stage and to provide appropriate treatment accordingly. • To provide special care to the women who have history of previous spontaneous abortion or preterm labour. • Advice the pregnant women regarding: Not to smoke and use of alcohol To take well balanced diet Avoid hard work Iron folic acid supplementation Maintaining adequate personal and environmental hygiene To contact the doctor immediately if any abnormal symptoms arise 2) Prevention at secondary level  In this level, actions are taken at the incipient stage of the disease so that it can be diagnosed.

PRETERM LABOR.pptx FOR NURSING STUDENTS CREATED BY KIRAN KARETHAKIRAN KARETHA

40 slides•52 views

PREMATURE LABOUR/ PRETERM LABOUR DEFINITION:  Pre term labour is defined by WHO as onset of labour prior to the completion of 37 weeks of gestation in a pregnancy beyond 20 weeks of gestation. INCIDENCE:  Globally, approximately 1 in 10 babies are born preterm (before 37 completed weeks of gestation), with an estimated 13.4 million preterm births annually. This means the incidence of preterm birth is roughly 10-11% worldwide.  In India, about 12% of babies are born preterm, which is higher than neighboring countries. ETIOLOGY:  In about 50%, the cause of preterm labour is not known- IDIOPATHIC  Some of the high-risk factors are:  HISTORY: previous history of abortion or preterm delivery Recurrent UTI Smoking habits Low socio-economic & nutritional status Previous abortion history Malpresentation  COMPLICATIONS IN PRESENT PREGNANCY: it may be due to maternal, fetal and placental. SIGN AND SYMPTOMS:  Backache  Contractions every 10 minutes, are more often  Cramping in lower abdomen  Menstrual like cramps  Fluid leaking from vagina  Flu like symptoms  Increased pressure in pelvis  Increased vaginal bleeding  Regular uterine activity  Vaginal spotting DIAGNOSIS:  Regular uterine contractions with or without pain (at least one in every 10 minutes.)  Dilation (2 cm or more) & effacement (80%) of the cervix  Length of cervix (2.5 cm or more.)  Funneling of internal OS INVESTIGATIONS:  Blood test  Urine analysis, urine culture and sensitivity  Cervicovaginal swab: culture and fibronectin test  Serum electrolyte and glucose level  USG COMPLICATION:  Birth of a pre-term baby A pre-term baby usually has following problems: • Low birth weight • Birth asphyxia • Neonatal jaundice • Underdeveloped organs etc. PREVENTIVE MEASURES: However, it is not possible to prevent occurrence of preterm labour completely, though its prevalence can be reduced using various measures at following three levels- 1) Prevention at primary level 2) Prevention at secondary level 3) Prevention at tertiary level 1) Prevention at primary level  In this, actions are taken prior to the onset of any disease which reduce or remove the possibility for occurring a disease.  For preventing preterm labour, following measures are applied at primary level. • Encourage the pregnant, to visit antenatal clinic regularly. • To identify high risk factors at its incipient stage and to provide appropriate treatment accordingly. • To provide special care to the women who have history of previous spontaneous abortion or preterm labour. • Advice the pregnant women regarding: Not to smoke and use of alcohol To take well balanced diet Avoid hard work Iron folic acid supplementation Maintaining adequate personal and environmental hygiene To contact the doctor immediately if any abnormal symptoms arise 2) Prevention at secondary level  In this level, actions are taken at the incipient stage of the disease so that it can be diagnosed.